Summary
- Splice variants
- Transcript comparison
- Gene alleles
Sequence
- Secondary Structure
Comparative Genomics
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- Gene gain/loss tree
- Orthologues
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Ontologies
- GO: Anatomical entity
Phenotypes
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Gene: PNISR ENSBTAG00000019730

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Description

PNN interacting serine and arginine rich protein [Source:UniProtKB Gene Name;Acc:F1N5A3]

Location

Primary_assembly 9: 50,357,664-50,375,219 forward strand.

ARS-UCD2.0:CM008176.2

About this gene

This gene has 2 transcripts (splice variants) and 166 orthologues.

Transcripts

Show transcript tableHide transcript table

Transcript ID	Name	bp	Protein	Translation ID	Biotype	UniProt Match	Flags
ENSBTAT00000026293.8	PNISR-201	2780	814aa	ENSBTAP00000026293.6	Gene/transcipt that contains an open reading frame (ORF).Protein coding	F1N5A3	A single transcript chosen for a gene which is the most conserved, most highly expressed, has the longest coding sequence and is represented in other key resources, such as NCBI and UniProt. This is defined in detail on http://www.ensembl.org/info/genome/genebuild/canonical.htmlEnsembl Canonical, APPRIS P2: Where the APPRIS core modules are unable to choose a clear principal variant (approximately 25% of human protein coding genes), the database chooses two or more of the CDS variants as "candidates" to be the principal variant. APPRIS is a system to annotate alternatively spliced transcripts based on a range of computational methods to identify the most functionally important transcript(s) of a gene. APPRIS P2,
ENSBTAT00000086229.3	PNISR-202	5130	805aa	ENSBTAP00000068846.2	Gene/transcipt that contains an open reading frame (ORF).Protein coding	A0A3Q1NBW7	APPRIS ALT2: For genes in which the APPRIS core modules are unable to choose a clear principal isoform, the ALT1 is the candidate transcript(s) models that appear to be conserved in fewer than three tested species. APPRIS is a system to annotate alternatively spliced transcripts based on a range of computational methods to identify the most functionally important transcript(s) of a gene. APPRIS ALT2,

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Summary

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